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Contact Us

Moscow

Department of Biological Sciences
biosci@uidaho.edu
phone: (208) 885-6280
fax:(208) 885-7905
Life Sciences South 252
875 Perimeter Drive MS 3051
Moscow, ID 83844-3051

Dr. Peter Fuerst

Peter G. Fuerst, Ph.D.


Office: LSS 141
Email: fuerst@uidaho.edu
Mailing Address: University of Idaho
Dept. of Biological Sciences
P. O. Box 443051
Moscow, ID 83844-3051

College of Science
Biological Sciences
Assistant Professor

Campus Locations: Moscow
With UI Since 2010


  • Research/Focus Areas
    • Neuroscience
    • Developmental Biology
    • Cell and Molecular Biology
    • Genetics
  • Selected Publications
    • Blank M, Fuerst, P.G., Stevens B, Nouri N, Kirkby L, Warrier D, Barres BA, Feller MB, Huberman AD, Burgess RW, Garner CC. (2011). The Down Syndrome Critical Region Regulates Retinogeniculate Refinement. J Neurosci. 2011 Apr 13;31 (15):5764-5776.
    • Fuerst, P. G., Harris, B.S., Johnson, K.R., and Burgess, R.W. (2010). A novel null allele of mouse Dscam survives to adulthood on an inbred C3H background with reduced phenotypic variability. Genesis, 2010 Oct 1;48(10):578-84.
    •  Fuerst, P.G., Bruce, F., Tian, M., Wei, W., Elstrott, J., Feller, M.B., Erskine, L., Singer, J.H., and Burgess, R.W. (2009). DSCAM and DSCAML1 function in self-avoidance in multiple cell types in the developing mouse retina.  Neuron 64, 484-497.
    •  Fuerst, P.G., and Burgess R.W. (2009).  Adhesion molecules in establishing retinal circuitry.  Current Opinion in Neurobiology 19, 389-394.
    • Fuerst, P.G., Koizumi, A., Masland, R.H., and Burgess, R.W. (2008). Neurite arborization and mosaic spacing in the mouse retina require DSCAM. Nature 451, 470-474.
    • Brady, T.L., Fuerst, P.G., Dick, R.A., Schmidt, C., and Voytas, D.F. (2008). Retrotransposon target site selection by imitation of a cellular protein. Molecular and Cellular Biology 28, 1230-1239.
    • Fuerst, P.G., Rauch, S.M., and Burgess, R.W. (2007). Defects in eye development in transgenic mice overexpressing the heparan sulfate proteoglycan agrin. Developmental Biology 303, 165-180.
    • Fuerst, P.G., and Voytas, D.F. (2003). CEN plasmid segregation is destabilized by tethered determinants of Ty 5 integration specificity: a role for double-strand breaks in CEN antagonism. Chromosoma 112, 58-65.
  • Research Projects
    • Genetic Regulation of Retinal Development: In the Fuerst lab, we use mouse models that have mutations or are transgenic to understand how the nervous system is wired, with a focus on the retina, the neural portion of the eye. The cells of the retina are roughly divided into cells that collect light information, the photoreceptors, interneurons that process information and the ganglion cells that send information to the rest of the brain. Our lab is interested in understanding how genes, and the proteins they encode, facilitate synaptic connectivity within the retina and brain in general.
    • Cell-Cell Interactions and the Balance of Life and Death: During development a large excess of neurons is produced. Neurons that integrate into functional circuitry survive while others undergo a process called developmental, or programmed, cell death. The balance between life and death for these neurons involve a type of protein on their surface, called cell adhesion molecules. We study how these molecules result in survival or death of cells by studying tissue in culture or with mouse models.
    • Neural Connectivity: The Human nervous system has an estimated 10,000 distinct types of neurons. Each of these neuron types makes a limited number of connections with a small number of other cell types. Our lab is interested in understanding how cell types choose the correct cells from which to receive information and to which to send information.

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