
Research/Focus Areas
- Molecular Parasitology
- Cell Biology
- Genetics
- Microbiology
My Courses
- MED 512: Cellular Physiology
- MMBB 463/563: Molecular Parasitology
- MMBB 589 Advanced Topics in Microbiology, Molecular Biology and Biochemistry
Gustavo Arrizabalaga, Ph.D.
College of Science
Biological Sciences
Associate Professor
Campus Locations
Moscow
With UI Since
2004
Office: LSS 145
Phone: (208) 885-6079
Email: gustavo@uidaho.eduMailing Address:University of Idaho
Dept. of Biological Sciences
P. O. Box 443051
Moscow, ID 83844-3051
- Ph.D., Massachusetts Institute of Technology, 1999
- B.S., Haverford College, 1992
Selected Publications
- Kamau E., Meehan T., Lavine M.D. Arrizabalaga, G., Mustata, G. and Boyle, J. 2011. A novel benzodioxole-containing inhibitors of Toxoplasma gondii growth alters the parasite cell cycle. Antimicrob Agents Chemother, 55(2): 745-55
- Francia, M.E., Wicher S., Pace D.A., Sullivan, J., Moreno, S.N.J., and Arrizabalaga, G. 2011. A Toxoplasma protein with homology to intracellular type Sodium Hydrogen Exchangers is required for osmotolerance and protein processing. Experimental Cell Research. 317(10):1382-96
- Arrizabalaga G. 2011. A fine balance between life and death: modulation of BCL-2 family members by Toxoplasma gondii. Frontier in Cellular and Infection Microbiology. 2:39
- Lavine, M.D. and Arrizabalaga, G. 2011. The antibiotic monensin causes cell cycle disruption of Toxoplasma gondii, mediated through the DNA repair enzyme TgMSH-1. Antimicrob Agents Chemother, 55(2): 745-55
- Garrison, E. and Arrizabalaga, G., 2009. Disruption of a mitochondrial homolog of a MutS DNA Repair Enzyme confers drug resistance in the pathogenic parasite Toxoplasma gondii. Molecular Microbiology, 72(2):425-41
- Lavine, M.D. and Arrizabalaga, G., 2009. Induction of mitotic S-phase of host and neighboring cells by Toxoplasma gondii enhances parasite invasion. Molecular and Biochemical Parasitology, 164(1):95-99
- Saeij, J., Arrizabalaga, G. and Boothroyd J.C., 2008. A cluster of four surface antigen genes specifically expressed in bradyzoites, SAG2CDXY, plays an important role in Toxoplasma gondii persistence. Immunity and infection, 76(6):2402-10
- Lavine, M and Arrizabalaga, G., 2008 Exit from host cells by the pathogenic parasite Toxoplasma gondii does not require motility. Eukaryotic Cell, 7(1): 131-40
Research Projects
- Analysis of the role of a MutS homolog in drug sensitivity in Toxoplasma gondii
- Identification of genes involved in calcium signaling in the parasite Toxoplasma gondii
- Genetic analysis of the regulation of motility in T. gondii.
- Use of T. gondii antigens as anti-asthma vaccines
Outreach Projects
- Grant Reviewer, American Cancer Society
Awards and Honors
- 2011 Commitment to Diversity Faculty Award, WWAMI medical program, University of Idaho
- 2011 Excellence in Teaching Award, WWAMI medical program, University of Idaho
- 2010 R.M. Wade Teaching Excellence Award, College of Agriculture and Life